You can download a Portable Document Format (PDF) version of the MAST Rating Form (198K). You can also download the MAST Written Commands (37K) and the Verbal Fluency Subtest Stimulus (63K). (What is PDF and How do I use PDF Files?) Copyright 1998-2012 Home Background Scales Survey Newsletter
Mast pdf free download
The Flag of the United States of America is a symbol of freedom and liberty to which Americans pledge their allegiance. Standing at attention and facing the flag with their right hand over the heart, they recite:
The United States flag flies at half-staff (or half-mast) when the nation or a state is in mourning. The president, through a presidential proclamation, a state governor, or the mayor of the District of Columbia can order flags to fly at half-staff. Most often, this is done to mark the death of a government official, military member, or first responder; in honor of Memorial Day or other national day of remembrance; or following a national tragedy.
The principal sensation of a mast is this permanent enjoyment of divine intoxication. The creation is full of bliss and the mast enjoys this bliss and thereby becomes intoxicated to an almost unlimited extent, virtually consuming him and absorbing him and thereby making the world around him vanish. Absorbed in God, such a person is continually absorbed in thinking about God, and with that comes like a bolt of pure love consuming him further in a state of divine intoxication.
There are those who have become masts whose minds have become unbalanced through unceasing dwelling upon thoughts of God so that they neglect all normal human requirements. There are those whose minds have become unbalanced by sudden contact with a highly advanced spiritual being. There are those who have sought spiritual experience and have met a crisis from which they do not recover. What characterizes all masts is their concentration upon the love of God. (pp 2031 Lord Meher Vol. 6)
The seven volumes span Sousa's entire march-writing career, from 1873-1932, and offer free resources for 129 marches. Modern recordings, historical information (courtesy of Paul E. Bierley, author of The Works of John Philip Sousa), as well as full-band scores and sheet music for marches that are in the public domain, are all available for public use as a result of this multi-year project. The volumes are available for free download exclusively on the Marine Band website.
Mast cell activation disease comprises disorders characterized by accumulation of genetically altered mast cells and/or abnormal release of these cells' mediators, affecting functions in potentially every organ system, often without causing abnormalities in routine laboratory or radiologic testing. In most cases of mast cell activation disease, diagnosis is possible by relatively non-invasive investigation. Effective therapy often consists simply of antihistamines and mast cell membrane-stabilising compounds supplemented with medications targeted at specific symptoms and complications. Mast cell activation disease is now appreciated to likely be considerably prevalent and thus should be considered routinely in the differential diagnosis of patients with chronic multisystem polymorbidity or patients in whom a definitively diagnosed major illness does not well account for the entirety of the patient's presentation.
MCAD is first suspected on clinical grounds, based on recognition of compatible mast cell mediator-related symptoms and, in some, identification of typical skin lesions. The clinical presentation of MCAD is very diverse, since due to both the widespread distribution of mast cells and the great heterogeneity of aberrant mediator expression patterns, symptoms can occur in virtually all organs and tissues (Table 3). Moreover, symptoms often occur in a temporally staggered fashion, waxing and waning over years to decades. Symptoms often initially manifest during adolescence or even childhood or infancy but are recognized only in retrospect as MCAD-related. Clinical features and courses vary greatly and range from very indolent with normal life expectancy to highly aggressive with reduced survival times. Physical examination should include inspection for a large assortment of types of skin lesions, testing for dermatographism (Darier's sign), and palpating for hepatosplenomegaly and lymphadenopathy. A diagnostic algorithm is shown in Figure 1. Recognition of a mast cell mediator release syndrome, i.e. a pattern of symptoms caused by the unregulated increased release of mediators from mast cells, can be aided by use of a validated checklist [2, 11, 12, 33] which lists the complaint complexes to be considered. In addition to the detection of the characteristic clinical constellation of findings, it must be investigated whether levels of the mast cell-specific mediators tryptase, histamine, and heparin are elevated in the blood, whether the excretion of the histamine metabolite methylhistamine into the urine is increased, and whether mast cell activity-related eosinophilia, basophilia or monocytosis in the blood can be observed. Other useful markers fairly specific to mast cells include serum chromogranin A (in the absence of cardiac and renal failure, neuroendocrine cancer, and proton pump inhibitor use) and serum and urinary leukotriene and prostaglandin isoforms (e.g., leukotriene E4, prostaglandin D2, and prostaglandin 9α,11βPGF2). Together with a characteristic clinical presentation, abnormal markers can be of diagnostic, therapeutic and prognostic relevance. However, it remains unsettled whether demonstration of an elevation of mast cell activity markers is absolutely necessary for diagnosis of MCAD because (1) many conditions (e.g., degrading enzymes, complexing molecules, tissue pH) may attenuate or impede spill-over of exocytosed mediators from tissues into the blood, (2) only a handful of the more than 60 releasable mast cell mediators can be detected by routine commercial techniques, and (3) mediator release syndrome may be due to an amplification cascade of basophil, eosinophil, and general leukocyte activation induced by liberation of only a few mast cell mediators [34] which, again, may not be detectable by present techniques.
An aggressive course of MCAD is characterized and defined by organopathy caused by pathologic infiltration of various organs by neoplastic mast cells inducing an impairment of organ function. Organopathy due to mast cell infiltration is indicated by findings termed C-findings: (1) significant cytopenia(s); (2) hepatomegaly with impairment of liver function due to mast cell infiltration, often with ascites; (3) splenomegaly with hypersplenism; (4) malabsorption with hypoalbuminemia and weight loss; (5) life-threatening impairment of organ function in other organ systems; (6) osteolyses and/or severe osteoporosis with pathologic fractures. Urticaria pigmentosa-like skin lesions are usually absent. In contrast to MCL, the bone marrow smear shows fewer than 20% mast cells (reviewed in [2]). Mast cell infiltration with organomegaly but without end organ dysfunction (hepatomegaly, splenomegaly, lymphadenopathy, bone marrow alterations) is a B-finding and may occur in a subvariant of SM (smoldering SM) with high mast cell burden.
The cornerstone of therapy is avoidance of identifiable triggers for mast cell degranulation such as animal venoms, extremes of temperature, mechanical irritation, alcohol, or medications (e.g., aspirin, radiocontrast agents, certain anesthetic agents). Individual patients may have variable tolerance patterns and avoidance lists, but it also is not uncommon to have no identifiable, reliable triggers.
MCAD comprises disorders affecting functions in potentially every organ system by abnormal release of mediators from and/or accumulation of genetically altered mast cells. There is evidence that MCAD is a disorder with considerable prevalence and thus should be considered routinely in the differential diagnosis of patients with chronic multisystem polymorbidity of unknown cause. In most cases of MCAD, diagnosis is possible by relatively non-invasive investigation. Effective therapy often consists simply of antihistamines and mast cell membrane-stabilising compounds supplemented with medications targeted at specific symptoms and complications.
Mast Climbing Work Platforms (MCWPs), or mast climbers, are a type of construction elevating equipment used to perform work at height. Mast climbers are equipped with a powered drive unit for propelling the work platform up and down a vertical mast structure. As with any type of equipment used to accomplish work at height, safety hazards may be present. Such hazards can result in injury or death.
Training is necessary for anyone using mast climber equipment. NIOSH has developed an online tool to help those who might use mast climbers identify common hazards. This educational resource can also help employers, trainers, and safety and health professionals to prevent work-related falls. Using the inspection tool is not a substitute for OSHA training requirements for MCWPs.
Note: Every mast climbing work platform configuration and set-up is specific to the site. Always consult manufacturer specifications and guidelines for each specific set-up as they can vary from site to site. Mention of any company or product does not constitute endorsement by the National Institute for Occupational Safety and Health, Centers for Disease Control and Prevention.
This free daily inspection walkthrough tool allows mast climber users to navigate through what is commonly inspected during a pre-shift daily inspection. When prompted, click on the orange outlined section and the related inspection point will be displayed. Please note the pictures displayed are of a typical mast climbing work platform configuration and do not include all set-ups that may be present on site. 2ff7e9595c
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